화학공학소재연구정보센터
검색결과 : 6건
No. Article
1 Rebound pathway overactivation by cancer cells following discontinuation of PI3K or mTOR inhibition promotes cancer cell growth
Faes S, Santoro T, Troquier L, Silva OD, Dormond O
Biochemical and Biophysical Research Communications, 513(3), 546, 2019
2 PI3K and AKT: Unfaithful Partners in Cancer
Faes S, Dormond O
International Journal of Molecular Sciences, 16(9), 21138, 2015
3 PGE(2)-induced colon cancer growth is mediated by mTORC1
Dufour M, Faes S, Dormond-Meuwly A, Demartines N, Dormond O
Biochemical and Biophysical Research Communications, 451(4), 587, 2014
4 Reactivation of AKT signaling following treatment of cancer cells with PI3K inhibitors attenuates their antitumor effects
Dufour M, Dormond-Meuwly A, Pythoud C, Demartines N, Dormond O
Biochemical and Biophysical Research Communications, 438(1), 32, 2013
5 The inhibition of MAPK potentiates the anti-angiogenic efficacy of mTOR inhibitors
Dormond-Meuwly A, Roulin D, Dufour M, Benoit M, Demartines N, Dormond O
Biochemical and Biophysical Research Communications, 407(4), 714, 2011
6 mTORC2 regulates PGE(2)-mediated endothelial cell survival and migration
Dada S, Demartines N, Dormond O
Biochemical and Biophysical Research Communications, 372(4), 875, 2008